Rick Shelton

Richard Shelton is the Chief Executive Officer and Chief Medical Officer for Centerstone Research Institute. He attended medical school at the University of Louisville in Kentucky. He became a resident and Chief Resident at the Massachusetts Mental Health Center (now the Longwood Program), a teaching hospital of the Harvard Medical School. After residency, he was a research fellow at the National Institutes of Mental Health Intramural Program in Washington, D.C. before entering the faculty of Vanderbilt University in 1985. There he became the James G. Blakemore Research Professor and Vice Chair for Clinical Research in the Department of Psychiatry. He also became a professor in the Departments of Psychology and Pharmacology.

Shelton joined the Department of Psychiatry and Behavioral Neurobiology at the University of Alabama at Birmingham in February 2012 as the Charles Byron Ireland Professor, Vice Chair for Research, and head of the Mood Disorders Research Program. He has served on NIH study sections and was the chair of the Adult Psychopathology and Disorders of Aging review group. He has also actively served as a teacher and mentor; he has won the Excellence in Teaching Award from the residents of the Vanderbilt Department of Psychiatry four times and the residents of UAB Department of Psychiatry twice. He was recognized by the National Alliance for the Mentally Ill for his service to persons with serious and persistent mental illnesses with the Exemplary Psychiatrist Award in 1994 and 2012. He also received the Dorothea Dix Professional Service Award from the Tennessee Association of Mental Health Centers in 2007 in recognition of his impact on the practice of Psychiatry in the State of Tennessee. Dr. Shelton’s research focuses on the development of new treatments for depression and bipolar disorder (experimental therapeutics), including the identification of new targets for treatment, prevention of serious mental illnesses, testing novel therapies, and identifying biomarkers of both disease and treatment response.

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